Ultrafine particulate matter pollution and dysfunction of endoplasmic reticulum Ca2+ store: A pathomechanism shared with amyotrophic lateral sclerosis motor neurons?-Reference-Cited by-同舟云学术

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Ultrafine particulate matter pollution and dysfunction of endoplasmic reticulum Ca2+ store: A pathomechanism shared with amyotrophic lateral sclerosis motor neurons?

Published:2024-03 Issue: Volume:273 Page:116104
ISSN:0147-6513
Container-title:Ecotoxicology and Environmental Safety
language:en
Short-container-title:Ecotoxicology and Environmental Safety

Author:

Sapienza Silvia,Tedeschi Valentina,Apicella Barbara,Pannaccione Anna,Russo Carmela,Sisalli Maria Josè,Magliocca Giorgia,Loffredo Stefania,Secondo Agnese

Funder

University of Naples Federico II

Publisher

Elsevier BV

Reference51 articles.

1. TRPC1, Orai1, and STIM1 in SOCE: friends in tight spaces;Ambudkar;Cell Calcium,2017

2. Structure-function relationships of the calcium binding sites of the sarcoplasmic reticulum Ca(2+)-ATPase;Andersen;Acta Physiol. Scand. Suppl.,1998

3. Prolonged NCX activation prevents SOD1 accumulation, reduces neuroinflammation, ameliorates motor behavior and prolongs survival in a ALS mouse model;Anzilotti;Neurobiol. Dis.,2021

4. PAHs and fullerenes as structural and compositional motifs tracing and distinguishing organic carbon from soot;Apicella;Fuel,2022

5. Distribution and isoform diversity of the organellar Ca2+ pumps in the brain;Baba-Aissa;Mol. Chem. Neuropathol.,1998

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