Interaction between nitric oxide signaling and gap junctions: Effects on vascular function-Reference-Cited by-同舟云学术

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Interaction between nitric oxide signaling and gap junctions: Effects on vascular function

Published:2012-08 Issue:8 Volume:1818 Page:1895-1902
ISSN:0005-2736
Container-title:Biochimica et Biophysica Acta (BBA) - Biomembranes
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Biomembranes

Author:

Looft-Wilson R.C.,Billaud M.,Johnstone S.R.,Straub A.C.,Isakson B.E.

Funder

NIH

AHA SDG

AHA post-doctoral fellowship

NRSA post-doctoral fellowship

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry,Biophysics

Reference111 articles.

1. Regulation of Nitric Oxide-Sensitive Guanylyl Cyclase

2. Cyclic guanosine monophosphate as a mediator of vasodilation.

3. An endothelium-derived hyperpolarizing factor distinct from NO and prostacyclin is a major endothelium-dependent vasodilator in resistance vessels of wild-type and endothelial NO synthase knockout mice

4. Cellular signaling and NO production

5. Nitric oxide synthase generates nitric oxide locally to regulate compartmentalized protein S-nitrosylation and protein trafficking

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