Genetic ablation of phosphatidylcholine transfer protein/StarD2 in ob/ob mice improves glucose tolerance without increasing energy expenditure
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Reference18 articles.
1. PC-TP/StARD2: of membranes and metabolism;Kang;Trends Endocrinol Metab,2010
2. Regulation of energy substrate utilization and hepatic insulin sensitivity by phosphatidylcholine transfer protein/StarD2;Scapa;FASEB J,2008
3. Genetic ablation or chemical inhibition of phosphatidylcholine transfer protein attenuates diet-induced hepatic glucose production;Shishova;Hepatology,2011
4. Mice lacking Pctp/StarD2 exhibit increased adaptive thermogenesis and enlarged mitochondria in brown adipose tissue;Kang;J Lipid Res,2009
5. Leptin and the regulation of body weight in mammals;Friedman;Nature,1998
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1. Ligand dependent interaction between PC-TP and PPARδ mitigates diet-induced hepatic steatosis in male mice;Nature Communications;2023-05-12
2. Evolutionary relationship analysis of STARD gene family and VvSTARD5 improves tolerance of salt stress in transgenic tomatoes;Physiologia Plantarum;2022-09
3. iTRAQ‐ and LC–MS/MS‐based quantitative proteomics reveals Pqlc2 as a potential regulator of hepatic glucose metabolism and insulin signalling pathway during fasting;Clinical and Experimental Pharmacology and Physiology;2020-11-03
4. The START-domain proteins in intracellular lipid transport and beyond;Molecular and Cellular Endocrinology;2020-03
5. Phosphatidylcholine transfer protein/StarD2 promotes microvesicular steatosis and liver injury in murine experimental steatohepatitis;American Journal of Physiology-Gastrointestinal and Liver Physiology;2017-07-01
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