Downregulation of Bit1 expression promotes growth, anoikis resistance, and transformation of immortalized human bronchial epithelial cells via Erk activation-dependent suppression of E-cadherin-Reference-Cited by-同舟云学术

Downregulation of Bit1 expression promotes growth, anoikis resistance, and transformation of immortalized human bronchial epithelial cells via Erk activation-dependent suppression of E-cadherin

Published:2018-01 Issue:1 Volume:495 Page:1240-1248
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Yao Xin,Gray Selena,Pham Tri,Delgardo Mychael,Nguyen An,Do Stephen,Ireland Shubha Kale,Chen Renwei,Abdel-Mageed Asim B.,Biliran Hector

Funder

Louisiana Cancer Research Consortium

NIH

NIH RCMI

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference21 articles.

1. A mitochondrial protein, Bit1, mediates apoptosis regulated by integrins and Groucho/TLE corepressors;Jan;Cell,2004

2. Protein kinase D is a positive regulator of Bit1 apoptotic function;Biliran;J. Biol. Chem.,2008

3. Anoikis effector Bit1 negatively regulates Erk activity;Kairouz-Wahbe;Proc. Natl. Acad. Sci. U. S. A.,2008

4. Karmali PP, Brunquell C, Tram H, Ireland SK, Ruoslahti E, Biliran H. Metastasis of tumor cells is enhanced by downregulation of Bit1. PLoS One 6: e23840.

5. Brunquell C, Biliran H, Jennings S, Ireland SK, Chen R, Ruoslahti E. TLE1 is an anoikis regulator and is downregulated by Bit1 in breast cancer cells. Mol. Cancer Res. 10: 1482–1495.

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