TRIF promotes angiotensin II-induced cross-talk between fibroblasts and macrophages in atrial fibrosis-Reference-Cited by-同舟云学术

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TRIF promotes angiotensin II-induced cross-talk between fibroblasts and macrophages in atrial fibrosis

Published:2015-08 Issue:1 Volume:464 Page:100-105
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Chen Xiao-Qing,Zhang Dao-Liang,Zhang Ming-Jian,Guo Meng,Zhan Yang-Yang,Liu Fang,Jiang Wei-Feng,Zhou Li,Zhao Liang,Wang Quan-Xing,Liu Xu

Funder

National Nature Science Foundation of China

Shanghai Jiao Tong University School of Medicine

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference29 articles.

1. Atrial fibrillation pathophysiology: implications for management;Iwasaki;Circulation,2011

2. Atrial remodeling and atrial fibrillation: recent advances and translational perspectives;Nattel;J. Am. Coll. Cardiol.,2014

3. Role of inflammation in initiation and perpetuation of atrial fibrillation: a systematic review of the published data;Issac;J. Am. Coll. Cardiol.,2007

4. Recruitment of immune cells across atrial endocardium in human atrial fibrillation;Yamashita;Circ. J.,2010

5. Pioglitazone inhibits angiotensin II-induced atrial fibroblasts proliferation via NF-κB/TGF-β1/TRIF/TRAF6 pathway;Chen;Exp. Cell. Res.,2015

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