All G protein βγ complexes are capable of translocation on receptor activation-Reference-Cited by-同舟云学术

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All G protein βγ complexes are capable of translocation on receptor activation

Published:2012-05 Issue:3 Volume:421 Page:605-611
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Ajith Karunarathne W.K.,O’Neill Patrick R.,Martinez-Espinosa Pedro L.,Kalyanaraman Vani,Gautam N.

Funder

NIH Grants

NRSA post doctoral fellowship

NIH grant

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference20 articles.

1. G protein pathways;Neves;Science,2002

2. Differential distribution of alpha subunits and beta gamma subunits of heterotrimeric G proteins on Golgi membranes of the exocrine pancreas;Denker;J. Cell. Biol.,1996

3. G protein beta interacts with the glucocorticoid receptor and suppresses its transcriptional activity in the nucleus;Kino;J. Cell. Biol.,2005

4. Activation of the phosphatidylinositol 3-kinase Vps34 by a G protein alpha subunit at the endosome;Slessareva;Cell,2006

5. G protein-coupled receptor-promoted trafficking of Gbeta1gamma2 leads to AKT activation at endosomes via a mechanism mediated by Gbeta1gamma2-Rab11a interaction;Garcia-Regalado;Mol. Biol. Cell,2008

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