Muscle spindles exhibit core lesions and extensive degeneration of intrafusal fibers in the Ryr1 mouse model of core myopathy
Author:
Funder
Canadian Institutes of Health Research
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference19 articles.
1. Core myopathies and malignant hyperthermia susceptibility: a review;Brislin;Paediatr. Anaesth.,2013
2. 150th ENMC International Workshop: Core Myopathies, 9–11th March 2007;Jungbluth,2008
3. Congenital hypotonia revisited;Brooke;Muscle Nerve,1979
4. Mechanistic models for muscle diseases and disorders originating in the sarcoplasmic reticulum;MacLennan;Biochim. Biophys. Acta,2011
5. Ca2+ dysregulation in Ryr1I4895T/wt mice causes congenital myopathy with progressive formation of minicores, cores, and nemaline rods;Zvaritch;Proc. Natl. Acad. Sci. U S A.,2009
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1. Evaluating Sexual Dimorphism of the Muscle Spindles and Intrafusal Muscle Fibers in the Medial Gastrocnemius of Male and Female Rats;Frontiers in Neuroanatomy;2021-10-01
2. Depletion of resident muscle stem cells negatively impacts running volume, physical function, and muscle fiber hypertrophy in response to lifelong physical activity;American Journal of Physiology-Cell Physiology;2020-06-01
3. Preclinical model systems of ryanodine receptor 1-related myopathies and malignant hyperthermia: a comprehensive scoping review of works published 1990–2019;Orphanet Journal of Rare Diseases;2020-05-07
4. The effects of neuregulin-1β on intrafusal muscle fiber formation in neuromuscular coculture of dorsal root ganglion explants and skeletal muscle cells;Skeletal Muscle;2018-09-15
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