Bile acid regulates c-Jun expression through the orphan nuclear receptor SHP induction in gastric cells
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference18 articles.
1. The influence of bile reflux, gastric acid and Helicobacter pylori infection on gastric mucosal injury: severity and localization;Chen;Weichang Bingxue,2002
2. Gastric pathology in cholecystectomy patients: role of Helicobacter pylori and bile reflux;Zullo;J. Clin. Gastroenterol.,1998
3. Activation of muscarinic receptor signaling by bile acids: physiological and medical implications;Raufman;Dig. Dis. Sci.,2003
4. Bile reflux gastritis and intestinal metaplasia at the cardia;Dixon;Gut,2002
5. Bile reflux gastritis and Barrett’s oesophagus: further evidence of a role for duodenogastro-oesophageal reflux?;Dixon;Gut,2001
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1. The role of bile acid in intestinal metaplasia;Frontiers in Physiology;2023-02-20
2. 4-Methylumbelliferone Targets Revealed by Public Data Analysis and Liver Transcriptome Sequencing;International Journal of Molecular Sciences;2023-01-21
3. Mechanism of action of NF-κB related cell signaling pathways in progression of gastritis to carcinoma;World Chinese Journal of Digestology;2022-03-28
4. Activation of FXR promotes intestinal metaplasia of gastric cells via SHP‑dependent upregulation of the expression of CDX2;Oncology Letters;2018-03-23
5. Bile acids-mediated overexpression of MUC4 via FAK-dependent c-Jun activation in pancreatic cancer;Molecular Oncology;2016-04-29
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