High glucose modifies transient receptor potential canonical type 6 channels via increased oxidative stress and syndecan-4 in human podocytes
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
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4. Podocytespecific overexpression of wild type or mutant trpc6 in mice is sufficient to cause glomerular disease;Krall;PLoS ONE,2010
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