Human monocytes and macrophages express NADPH oxidase 5; a potential source of reactive oxygen species in atherosclerosis-Reference-Cited by-同舟云学术

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Human monocytes and macrophages express NADPH oxidase 5; a potential source of reactive oxygen species in atherosclerosis

Published:2015-05 Issue:1 Volume:461 Page:172-179
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Manea Adrian,Manea Simona-Adriana,Gan Ana Maria,Constantin Alina,Fenyo Ioana Madalina,Raicu Monica,Muresian Horia,Simionescu Maya

Funder

UEFISCDI

European Social Found

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference29 articles.

1. Local proliferation dominates lesional macrophage accumulation in atherosclerosis;Robbins;Nat. Med.,2013

2. Regulation of superoxide anion production by NADPH oxidase in monocytes/macrophages: contributions to atherosclerosis;Cathcart;Arterioscler. Thromb. Vasc. Biol.,2004

3. Macrophages generate reactive oxygen species in response to minimally oxidized low-density lipoprotein: toll-like receptor 4- and spleen tyrosine kinase-dependent activation of NADPH oxidase 2;Bae;Circ. Res.,2009

4. IRAK-1 contributes to lipopolysaccharide-induced reactive oxygen species generation in macrophages by inducing NOX-1 transcription and Rac1 activation and suppressing the expression of antioxidative enzymes;Maitra;J. Biol. Chem.,2009

5. Nox4 is a novel inducible source of reactive oxygen species in monocytes and macrophages and mediates oxidized low density lipoprotein-induced macrophage death;Lee;Circ. Res.,2010

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