Reactive oxygen species activate HIV long terminal repeat via post-translational control of NF-κB
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
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3. Hyper-responsiveness to stimulation HIV-infected CD4+ T cells requires Nef and Tat virus gene products and results from higher NFAT, NF-κB, and AP-1 induction;Fortin;J. Biol. Chem.,2004
4. HIV-1 Tat potentiates TNF-induced NF-κB activation and cytotoxicity by altering redox state;Westendorp;EMBO J.,1995
5. Transient exposure to HIV-1 Tat protein results in cytokine production in macrophages and astrocytes;Nath;J. Biol. Chem.,1999
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