Impairment of the extrusion transporter for asymmetric dimethyl-l-arginine: A novel mechanism underlying vasospastic angina-Reference-Cited by-同舟云学术

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Impairment of the extrusion transporter for asymmetric dimethyl-l-arginine: A novel mechanism underlying vasospastic angina

Published:2012-06 Issue:2 Volume:423 Page:218-223
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Closs Ellen I.,Ostad Mir A.,Simon Alexandra,Warnholtz Ascan,Jabs Alexander,Habermeier Alice,Daiber Andreas,Förstermann Ulrich,Münzel Thomas

Funder

Federal Ministry of Education and Research

Stiftung Mainzer Herz

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference18 articles.

1. Variantform of angina pectoris, previously undelineated syndrome;Prinzmetal;JAMA,1960

2. Endothelial function and coronary spastic angina;Kawano;Intern. Med.,2005

3. Local coronary supersensitivity to diverse vasoconstrictive stimuli in patients with variant angina;Kaski;Circulation,1986

4. Spontaneous coronary artery spasm in variant angina is caused by a local hyperreactivity to a generalized constrictor stimulus;Kaski;J. Am. Coll. Cardiol.,1989

5. Nitric oxide-mediated flow-dependent dilation is impaired in coronary arteries in patients with coronary spastic angina;Kugiyama;J. Am. Coll. Cardiol.,1997

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