Cystathionine β-synthase is enriched in the brains of Down’s patients-Reference-Cited by-同舟云学术

Cystathionine β-synthase is enriched in the brains of Down’s patients

Published:2005-12 Issue:3 Volume:338 Page:1547-1550
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Ichinohe Akiko,Kanaumi Takeshi,Takashima Sachio,Enokido Yasushi,Nagai Yasuo,Kimura Hideo

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference18 articles.

1. Occurrence of neuropathological changes and dementia of Alzheimer’s disease in Down’s syndrome;Wisniewski;Ann. Neurol.,1985

2. Disorders of transsulfuration;Mudd,1989

3. Expression of chromosome 21-localized genes in acute myeloid leukemia: differences between Down syndrome and non-Down syndrome blast cells and relationship to in vitro sensitivity to cytosine arabinoside and daunorubicin;Taub;Blood,1999

4. Polymorphisms of genes controlling homocysteine/folate metabolism and cognitive function;Barbaux;Neuroreport,2000

5. Characterization of the enzymic capacity for cysteine desulphydration in liver and kidney of the rat;Stipanuk;Biochem. J.,1982

Cited by 113 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Neurobehavioral dysfunction in a mouse model of Down syndrome: upregulation of cystathionine β-synthase, H2S overproduction, altered protein persulfidation, synaptic dysfunction, endoplasmic reticulum stress, and autophagy;GeroScience;2024-04-01

2. Hydrogen Sulfide (H2S)/Polysulfides (H2Sn) Signalling and TRPA1 Channels Modification on Sulfur Metabolism;Biomolecules;2024-01-19

3. Oxidation of Hydrogen Sulfide to Polysulfide and Thiosulfate by a Carbon Nanozyme: Therapeutic Implications with an Emphasis on Down Syndrome;Advanced Materials;2023-07-23

4. Protective Roles of Hydrogen Sulfide in Alzheimer’s Disease and Traumatic Brain Injury;Antioxidants;2023-05-13

5. Advances of H2S in Regulating Neurodegenerative Diseases by Preserving Mitochondria Function;Antioxidants;2023-03-06