CIRP downregulation renders cardiac cells prone to apoptosis in heart failure
Author:
Funder
Wuhan University
National Natural Science Foundation of China
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference23 articles.
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2. Cold-inducible RNA-binding protein regulates cardiac repolarization by targeting transient outward potassium channels;Li;Circ. Res.,2015
3. The cold-inducible RNA-binding protein migrates from the nucleus to cytoplasmic stress granules by a methylation-dependent mechanism and acts as a translational repressor;De Leeuw;Exp. Cell Res.,2007
4. Cold-inducible proteins CIRP and RBM3, a unique couple with activities far beyond the cold;Zhu;Cell. Mol. Life Sci. : CMLS,2016
5. Cold-inducible RNA-binding protein (CIRP) triggers inflammatory responses in hemorrhagic shock and sepsis;Qiang;Nat. Med.,2013
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1. Cold inducible RNA binding protein-regulated mitochondria associated endoplasmic reticulum membranes-mediated Ca2+ transport play a critical role in hypothermia cerebral resuscitation;Experimental Neurology;2024-09
2. Inhibition of CIRBP represses the proliferation and migration of vascular smooth muscle cells via inhibiting Rheb/mTORC1 axis;Biochemical and Biophysical Research Communications;2024-09
3. Upregulation of CIRP by its agonist prevents the development of heart failure in myocardial infarction rats;BMC Cardiovascular Disorders;2024-03-27
4. Sphingosylphosphorylcholine alleviates pressure overload-induced myocardial remodeling in mice via inhibiting CaM-JNK/p38 signaling pathway;Acta Pharmacologica Sinica;2023-10-13
5. Targeting of cold-inducible RNA-binding protein alleviates sepsis via alleviating inflammation, apoptosis and oxidative stress in heart;International Immunopharmacology;2023-09
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