FGF-23 transgenic mice demonstrate hypophosphatemic rickets with reduced expression of sodium phosphate cotransporter type IIa-Reference-Cited by-同舟云学术

FGF-23 transgenic mice demonstrate hypophosphatemic rickets with reduced expression of sodium phosphate cotransporter type IIa

Published:2004-02 Issue:2 Volume:314 Page:409-414
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Shimada Takashi,Urakawa Itaru,Yamazaki Yuji,Hasegawa Hisashi,Hino Rieko,Yoneya Takashi,Takeuchi Yasuhiro,Fujita Toshiro,Fukumoto Seiji,Yamashita Takeyoshi

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference26 articles.

1. Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia;Shimada;Proc. Natl. Acad. Sci. USA,2001

2. The autosomal dominant hypophosphatemic rickets (ADHR) gene is a secreted polypeptide overexpressed by tumors that cause phosphate wasting;White;J. Clin. Endocrinol. Metab.,2001

3. The ADHR Consortium, Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23, Nat. Genet. 26 (2000) 345–348

4. Phosphorus homeostasis and related disorders;Drezner,1996

5. The autosomal dominant hypophosphatemic rickets R176Q mutation in FGF23 resists proteolytic cleavage and enhances in vivo biological potency;Bai;J. Biol. Chem.,2003

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