cIAPs promote the proteasomal degradation of mutant SOD1 linked to familial amyotrophic lateral sclerosis-Reference-Cited by-同舟云学术

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cIAPs promote the proteasomal degradation of mutant SOD1 linked to familial amyotrophic lateral sclerosis

Published:2016-11 Issue:3 Volume:480 Page:422-428
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Choi Jin Sun,Kim Kidae,Lee Do Hee,Cho Sayeon,Ha Jae Du,Park Byoung Chul,Kim Sunhong,Park Sung Goo,Kim Jeong-Hoon

Funder

Korea Research Institute of Bioscience and Biotechnology

National Research Foundation of Korea

National Research Council of Science and Technology

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference27 articles.

1. Ubiquitin, cellular inclusions and their role in neurodegeneration;Alves-Rodrigues;Trends Neurosci.,1998

2. Superoxide dismutase in familial amyotrophic lateral sclerosis: models for gain of function;Brown;Curr. Opin. Neurobiol.,1995

3. Enzyme function of copper, zinc superoxide dismutase as a free radical generator;Yim;J. Biol. Chem.,1993

4. A gain-of-function of an amyotrophic lateral sclerosis-associated Cu,Zn-superoxide dismutase mutant: an enhancement of free radical formation due to a decrease in Km for hydrogen peroxide;Yim;Proc. Natl. Acad. Sci. U. S. A.,1996

5. Proteasomal inhibition by misfolded mutant superoxide dismutase 1 induces selective motor neuron death in familial amyotrophic lateral sclerosis;Urushitani;J. Neurochem.,2002

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3. The biophysics of superoxide dismutase-1 and amyotrophic lateral sclerosis;Quarterly Reviews of Biophysics;2019

4. Promoting the clearance of neurotoxic proteins in neurodegenerative disorders of ageing;Nature Reviews Drug Discovery;2018-08-17

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