Lack of the P2X7 receptor protects against AMD-like defects and microparticle accumulation in a chronic oxidative stress-induced mouse model of AMD-Reference-Cited by-同舟云学术

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Lack of the P2X7 receptor protects against AMD-like defects and microparticle accumulation in a chronic oxidative stress-induced mouse model of AMD

Published:2017-01 Issue:1 Volume:482 Page:81-86
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Carver Kyle A.,Lin C.M.,Bowes Rickman Catherine,Yang Dongli

Funder

National Eye Institute

University of Michigan Start-Up Funds

NEI

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference32 articles.

1. Dry age-related macular degeneration: mechanisms, therapeutic targets, and imaging;Bowes Rickman;Invest. Ophthalmol. Vis. Sci.,2013

2. Retinal ultrastructure of murine models of dry age-related macular degeneration (AMD);Ramkumar;Prog. Retin Eye Res.,2010

3. Mechanisms of age-related macular degeneration and therapeutic opportunities;van Lookeren Campagne;J. Pathol.,2014

4. Consequences of oxidative stress in age-related macular degeneration;Jarrett;Mol. Asp. Med.,2012

5. Drusen, choroidal neovascularization, and retinal pigment epithelium dysfunction in SOD1-deficient mice: a model of age-related macular degeneration;Imamura;Proc. Natl. Acad. Sci. U. S. A.,2006

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