Disruption of Epac1 protects the heart from adenylyl cyclase type 5-mediated cardiac dysfunction-Reference-Cited by-同舟云学术

Disruption of Epac1 protects the heart from adenylyl cyclase type 5-mediated cardiac dysfunction

Published:2016-06 Issue:1 Volume:475 Page:1-7
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Cai Wenqian,Fujita Takayuki,Hidaka Yuko^ORCID,Jin Huiling,Suita Kenji,Prajapati Rajesh,Liang Chen,Umemura Masanari,Yokoyama Utako,Sato Motohiko,Okumura Satoshi,Ishikawa Yoshihiro

Funder

Japan Society for the Promotion of Science (JSPS) KAKENHI

Ministry of Education, Culture, Sports, Science and Technology (MEXT) KAKENHI

Strategic Research Foundation at Private Universities

New Energy and Industrial Technology Development Organization (NEDO)

National Cerebral and Cardiovascular Center (NCVC)

Japan Agency for Medical Research and Development (AMED)

Yokohama City University

Naito Foundation

Senshin Medical Research Foundation

Tokyo Biochemical Research Foundation

Academic Contribution from Pfizer Japan

Research Foundation for Community Medicine

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference39 articles.

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3. The human sympathetic nervous system: its relevance in hypertension and heart failure;Parati;Eur. Heart J.,2012

4. Disruption of type 5 adenylyl cyclase enhances desensitization of cyclic adenosine monophosphate signal and increases Akt signal with chronic catecholamine stress;Okumura;Circulation,2007

5. Epac1-dependent phospholamban phosphorylation mediates the cardiac response to stresses;Okumura;J. Clin. Investig.,2014

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