Myeloma cells self-promote migration by regulating TAB1-driven TIMP-1 expression in mesenchymal stem cells
Author:
Funder
National Medical Research Council
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference21 articles.
1. Modification of the bone marrow MSC population in a xenograft model of early multiple myeloma;L Berlier;Biochem. Biophys. Res. Commun.,2018
2. Mesenchymal stem cells inhibit breast cancer cell migration and invasion through secretion of tissue inhibitor of metalloproteinase-1 and -2;Clarke;Mol. Carcinog.,2015
3. MiR-29a reduces TIMP-1 production by dermal fibroblasts via targeting TGF-β activated kinase 1 binding protein 1, implications for systemic sclerosis;Ciechomska;PloS One,2014
4. Contact of myeloma cells induces a characteristic transcriptome signature in skeletal precursor cells –Implications for myeloma bone disease;Dotterweich;Bone,2016
5. Multiple myeloma in the marrow: pathogenesis and treatments;Fairfield;Ann. N. Y. Acad. Sci.,2016
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1. Characterization of the biological and transcriptomic landscapes of bone marrow-derived mesenchymal stem cells in patients with multiple myeloma;Cancer Cell International;2024-03-27
2. Matrix metalloproteinases and tissue inhibitors in multiple myeloma: promote or inhibit?;Frontiers in Oncology;2023-09-26
3. Myeloma Microenvironmental TIMP1 Induces the Invasive Phenotype in Fibroblasts to Modulate Disease Progression;International Journal of Molecular Sciences;2023-01-22
4. Construction of wound repair model and function of recombinant TIMP from Hyriopsis cumingii;Fish & Shellfish Immunology;2021-12
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