Overexpression of Dyrk1A regulates cardiac troponin T splicing in cells and mice-Reference-Cited by-同舟云学术

Overexpression of Dyrk1A regulates cardiac troponin T splicing in cells and mice

Published:2016-05 Issue:4 Volume:473 Page:993-998
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Lu Shu,Yin Xiaomin

Funder

Natural Science Foundation of Nantong University

National Natural Science Foundation of China

Basic Research Program of Jiangsu Education Department

Priority Academic Program Development of Jiangsu Higher Education Institution (PAPD)

The Open Program of Key Laboratory Foundation of Capital Medical University

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference26 articles.

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2. Molecular basis of human cardiac troponin T isoforms expressed in the developing, adult, and failing heart;Anderson;Circ. Res.,1995

3. Alternative RNA splicing-generated cardiac troponin T isoform switching: a non-heart-restricted genetic programming synchronized in developing cardiac and skeletal muscles;Jin;Biochem. Biophys. Res. Commun.,1996

4. A single cardiac troponin T gene generates embryonic and adult isoforms via developmentally regulated alternate splicing;Cooper;J. Biol. Chem.,1985

5. Cardiac troponin T isoforms affect the Ca(2+) sensitivity of force development in the presence of slow skeletal troponin I: insights into the role of troponin T isoforms in the fetal heart;Gomes;J. Biol. Chem.,2004

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