PP2Cα aggravates neuronal insulin resistance leading to AD-like phenotype in vitro-Reference-Cited by-同舟云学术

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PP2Cα aggravates neuronal insulin resistance leading to AD-like phenotype in vitro

Published:2023-02 Issue: Volume:644 Page:49-54
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Yadav Yamini,Dey Chinmoy Sankar

Funder

Council of Scientific and Industrial Research, India

Department of Science and Technology, Ministry of Science and Technology, India

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference26 articles.

1. Role of phosphatidylinositol 3-kinase activation on insulin action and its alteration in diabetic conditions;Asano;Biol. Pharm. Bull.,2007

2. Insulin in the brain: its pathophysiological implications for states related with central insulin resistance, type 2 diabetes and Alzheimer's disease;Blázquez;Front. Endocrinol.,2014

3. Brain control of blood glucose levels: implications for the pathogenesis of type 2 diabetes;Alonge;Diabetologia,2021

4. Glucose metabolism and insulin receptor signal transduction in Alzheimer disease;Hoyer;Eur. J. Pharmacol.,2004

5. Impaired insulin and insulin-like growth factor expression and signaling mechanisms in Alzheimer's disease--is this type 3 diabetes?;Steen;J Alzheimers Dis,2005

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1. Short‐Term Statin Therapy Induces Hepatic Insulin Resistance Through HNF4α/PAQR9/PPM1α Axis Regulated AKT Phosphorylation;Advanced Science;2024-07-05

2. HIF-1α serves as a co-linker between AD and T2DM;Biomedicine & Pharmacotherapy;2024-02

3. Neuronal insulin signaling and resistance: a balancing act of kinases and phosphatases;Journal of Endocrinology;2023-11-06

4. Novel insights into D-Pinitol based therapies: a link between tau hyperphosphorylation and insulin resistance;Neural Regeneration Research;2023-07-07

5. Recent Development in the Understanding of Molecular and Cellular Mechanisms Underlying the Etiopathogenesis of Alzheimer’s Disease;International Journal of Molecular Sciences;2023-04-14

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