Cardioprotective effects of constitutively active MEK1 against H2O2-induced apoptosis and autophagy in cardiomyocytes via the ERK1/2 signaling pathway-Reference-Cited by-同舟云学术

Cardioprotective effects of constitutively active MEK1 against H2O2-induced apoptosis and autophagy in cardiomyocytes via the ERK1/2 signaling pathway

Published:2019-04 Issue:1 Volume:512 Page:125-130
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Sun Ming-Hui,Chen Xiao-Cui,Han Min,Yang Yi-Ning,Gao Xiao-Ming,Ma Xiang,Huang Ying,Li Xiao-Mei,Gai Min-Tao,Liu Fen,Ma Yi-Tong,Chen Bang-Dang

Funder

Xinjiang Uygur Autonomous Region Science and Technology Department

Xinjiang Uygur Autonomous Region Ten-thousands Talents Program Backup Candidates

National Natural Science Foundation of China

Xinjiang Uygur Autonomous Region Graduate Research and Innovation

Xinjiang Medical University Graduate Innovation and Entrepreneurship

Tianshan Innovation Team

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

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4. H (2)O(2) opens mitochondrial K(ATP) channels and inhibits GABA receptors via protein kinase C-epsilon in cardiomyocytes;Zhang;Am. J. Physiol. Heart Circ. Physiol.,2002

5. GYY4137 protects against myocardial ischemia and reperfusion injury by attenuating oxidative stress and apoptosis in rats;Meng;J. Biomed. Res.,2015

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