Kaiso is a key regulator of spleen germinal center formation by repressing Bcl6 expression in splenocytes-Reference-Cited by-同舟云学术

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Kaiso is a key regulator of spleen germinal center formation by repressing Bcl6 expression in splenocytes

Published:2013-12 Issue:3-4 Volume:442 Page:177-182
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Koh Dong-In,Yoon Jae-Hyeon,Kim Min-Kyeong,An Haemin,Kim Min-Young,Hur Man-Wook

Funder

National Research Foundation (NRF)

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference19 articles.

1. A role for KAISO–p120ctn complexes in cancer?;Roy;Nat. Rev. Cancer,2005

2. N-CoR mediates DNA methylation-dependent repression through a methyl CpG binding protein KAISO;Yoon;Mol. Cell,2003

3. Specific peptide interference reveals BCL6 transcriptional and oncogenic mechanisms in B-cell lymphoma cells;Polo;Nat. Med.,2004

4. BCL-6 represses genes that function in lymphocyte differentiation, inflammation, and cell cycle control;Shaffer;Immunity,2000

5. Direct repression of prdm1 by Bcl-6 inhibits plasmacytic differentiation;Tunyaplin;J. Immunol.,2004

Cited by 9 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. A proposed novel genetic and epigenetic effects of Betulinic acid against breast cancer cells: in silico and in vitro study.;Egyptian Journal of Chemistry;2023-01-16

2. Deletion of murine Rhoh leads to de-repression of Bcl-6 via decreased KAISO levels and accelerates a malignancy phenotype in a murine model of lymphoma;Small GTPases;2022-01-05

3. Zbtb33 Gene Knockout Changes Transcription of the Fgf9, Fgfr3, c-Myc and FoxG1 Genes in the Developing Mouse Brain;Molecular Biology;2021-05

4. ZBTB33 (Kaiso) methylated binding sites are associated with primed heterochromatin;2019-03-24

5. Kaiso depletion attenuates the growth and survival of triple negative breast cancer cells;Cell Death & Disease;2017-03

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