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Drebrin is induced during myofibroblast differentiation and enhances the production of fibrosis-related genes

  • Published:2020-08 Issue:2 Volume:529 Page:224-230
  • ISSN:0006-291X
  • Container-title:Biochemical and Biophysical Research Communications
  • language:en
  • Short-container-title:Biochemical and Biophysical Research Communications

Author:

Hironaka Takanori,Ueno Tomoyuki,Mae Kyosuke,Yoshimura Chikashi,Morinaga Takumi,Horii Yuma,Nagasaka Akiomi,Kurose Hitoshi,Nakaya Michio

Funder

KAKENHI

Takeda Science Foundation

Mochida Memorial Foundation for Medical and Pharmaceutical Research

MSD Life Science Foundation

Senri Life Science Foundation

GSK Japan Research

Japan Agency for Medical Research and Development

Drug Discovery and Life Science Research

AMED

JSPS

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference26 articles.

1. Organ and tissue fibrosis: molecular signals, cellular mechanisms and translational implications;Weiskirchen;Mol. Aspect. Med.,2019

2. Fibrotic disease and the TH1/TH2 paradigm;Wynn;Nat. Rev. Immunol.,2004

3. The myofibroblast: one function, multiple origins;Hinz;Am. J. Pathol.,2007

4. TGFβ signaling and the control of myofibroblast differentiation: implications for chronic inflammatory disorders;Carthy;J. Cell. Physiol.,2018

5. Alpha-smooth muscle actin is transiently expressed by myofibroblasts during experimental wound healing;Darby;Lab. Invest.,1990

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