Hypoxia potentiates LPS-induced inflammatory response and increases cell death by promoting NLRP3 inflammasome activation in pancreatic β cells-Reference-Cited by-同舟云学术

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Hypoxia potentiates LPS-induced inflammatory response and increases cell death by promoting NLRP3 inflammasome activation in pancreatic β cells

Published:2018-01 Issue:4 Volume:495 Page:2512-2518
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Chen Cheng,Ma Xiaoqian,Yang Cejun,Nie Wei,Zhang Juan,Li Hongde,Rong Pengfei,Yi Shounan,Wang Wei

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference44 articles.

1. Regulation of insulin secretion: role of mitochondrial signalling;Jitrapakdee;Diabetologia,2010

2. The molecular mechanisms of pancreatic beta-cell glucotoxicity: recent findings and future research directions;Bensellam;Mol. Cell. Endocrinol.,2012

3. Low antioxidant enzyme gene expression in pancreatic islets compared with various other mouse tissues;Lenzen;Free Radic. Biol. Med.,1996

4. Glucose-induced O(2) consumption activates hypoxia inducible factors 1 and 2 in rat insulin-secreting pancreatic beta-cells;Bensellam;PLos One,2012

5. Acute hypoxia induces apoptosis of pancreatic beta-cell by activation of the unfolded protein response and upregulation of CHOP;Zheng;Cell Death Dis.,2012

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