Deletion of yeast TPK1 reduces the efficiency of non-homologous end joining DNA repair
Author:
Funder
Natural Sciences and Engineering Research Council of Canada
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference37 articles.
1. DNA double-strand-break complexity levels and their possible contributions to the probability for error-prone processing and repair pathway choice;Schipler;Nucleic Acids Res.,2013
2. Modernizing the nonhomologous end-joining repertoire: alternative and classical NHEJ share the stage;Deriano;Annu. Rev. Genet.,2013
3. Double-strand break end resection and repair pathway choice;Symington;Annu. Rev. Genet.,2011
4. Repair of double-strand breaks by end joining;K Chiruvella;Cold Spring Harb Perspect Biol,2013
5. Recruitment and dissociation of nonhomologous end joining proteins at a DNA double-strand break in saccharomyces cerevisiae;Wu;Genetics,2008
Cited by 5 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Isoform-specific sequestration of protein kinase A fine-tunes intracellular signaling during heat stress;Cell Reports;2024-06
2. Discovery and identification of genes involved in DNA damage repair in yeast;Gene;2022-07
3. Heat stress regulates the expression of TPK1 gene at transcriptional and post-transcriptional levels in Saccharomyces cerevisiae;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;2022-04
4. The conserved Tpk1 regulates non-homologous end joining double-strand break repair by phosphorylation of Nej1, a homolog of the human XLF;Nucleic Acids Research;2021-07-09
5. Multiple metabolic signals including AMPK and PKA regulate glucose-stimulated double strand break resection in yeast;2021-02-26
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