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High glucose downregulates the effects of autophagy on osteoclastogenesis via the AMPK/mTOR/ULK1 pathway

  • Published:2018-09 Issue:2 Volume:503 Page:428-435
  • ISSN:0006-291X
  • Container-title:Biochemical and Biophysical Research Communications
  • language:en
  • Short-container-title:Biochemical and Biophysical Research Communications

Author:

Cai Zhen-yu,Yang Bo,Shi Ying-xu,Zhang Wei-lin,Liu Fei,Zhao Wei,Yang Mao-WeiORCID

Funder

Chinese National Natural Science Foundation Project

Liaoning Province Department of Education

Liaoning Province Natural Science Foundation

Shenyang Municipal Science and Technology Fund

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference22 articles.

1. IDF Diabetes Atlas;International Diabetes Federation,2017

2. Diabetes Working, Mechanisms of diabetes mellitus-induced bone fragility;Napoli;J. Nat. Rev. Endocrinol.,2016

3. Diabetes and its effect on bone and fracture healing;Jiao;J. Curr. Osteoporos. Rep.,2015

4. High d(+)glucose concentration inhibits RANKL-induced osteoclastogenesis;Wittrant;J. Bone,2008

5. High glucose inhibits receptor activator of nuclear factorkappaB ligand-induced osteoclast differentiation via downregulation of vATPase V0 subunit d2 and dendritic cellspecific transmembrane protein;Xu;J. Mol. Med. Rep.,2015
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