Increased oxidative damage and mitochondrial abnormalities in the peripheral blood of Huntington’s disease patients-Reference-Cited by-同舟云学术

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Increased oxidative damage and mitochondrial abnormalities in the peripheral blood of Huntington’s disease patients

Published:2007-07 Issue:2 Volume:359 Page:335-340
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Chen Chiung-Mei,Wu Yih-Ru,Cheng Mei-Ling,Liu Jun-Liang,Lee Yu-May,Lee Po-Wei,Soong Bing-Wen,Chiu Daniel Tsun-Yee

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference32 articles.

1. HDCRG, The Huntington’s Disease Collaborative Research Group, A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington’s disease chromosomes, Cell 72 (1993) 971–983.

2. Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brain;DiFiglia;Science,1997

3. Therapeutics development for triplet repeat expansion diseases;Di Prospero;Nat. Rev. Genet.,2005

4. Increased apoptosis of Huntington disease lymphoblasts associated with repeat length-dependent mitochondrial depolarization;Sawa;Nat. Med.,1999

5. Autophagosome-like vacuole formation in Huntington’s disease lymphoblasts;Nagata;Neuroreport,2004

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