25-hydroxycholesterol promotes RANKL-induced osteoclastogenesis through coordinating NFATc1 and Sp1 complex in the transcription of miR-139-5p-Reference-Cited by-同舟云学术

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25-hydroxycholesterol promotes RANKL-induced osteoclastogenesis through coordinating NFATc1 and Sp1 complex in the transcription of miR-139-5p

Published:2017-04 Issue:4 Volume:485 Page:736-741
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Zhang Lishan,Lv Yinping,Xian Guozhe,Lin Yanliang

Funder

National Natural Science Foundation of China

Promotive research fund for excellent young and middle-aged scientists of Shandong Province

Medical and Health Technology Development Plan Project of Shandong Province

Science and Technology Development Plan Project of Shandong Province

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference26 articles.

1. NFAT and Osterix cooperatively regulate bone formation;Koga;Nat. Med.,2005

2. Suppression of RANKL-induced osteoclast differentiation by cilostazol via SIRT1-induced RANK inhibition;Park;Biochim. Biophys. Acta,2015

3. ATF3 modulates calcium signaling in osteoclast differentiation and activity byassociating with c-Fos and NFATc1 proteins;Jeong;Bone,2017

4. The hematopoietic transcription factor PU.1 regulates RANK gene expression inmyeloid progenitors;Kwon;Biochem. Biophys. Res. Commun.,2005

5. Induction and activation of the transcription factor NFATc1 (NFAT2) integrateRANKL signaling in terminal differentiation of osteoclasts;Takayanagi;Dev. Cell,2002

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4. 25-Hydroxycholesterol-induced Osteoblast Oxiapoptophagy Is Involved in the Pathophysiological Process of Osteoporosis;In Vivo;2023

5. 25‐Hydroxycholesterol induces odontoclastic differentiation through RANK – RANKL upregulation and NF‐κB activation in odontoblast‐like MDPC ‐23 cells: An in vitro study;International Endodontic Journal;2022-12-12

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