The sclerostin-bone protein interactome
Author:
Funder
NIH Grant
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference52 articles.
1. Osteocyte control of bone formation via sclerostin, a novel BMP antagonist;Winkler;EMBO J.,2003
2. Increased bone density in sclerosteosis is due to the deficiency of a novel secreted protein (SOST);Balemans;Hum. Mol. Genet.,2001
3. Identification of a 52 kb deletion downstream of the SOST gene in patients with van Buchem disease;Balemans;J. Med. Genet.,2002
4. Bone dysplasia sclerosteosis results from loss of the SOST gene product, a novel cystine knot-containing protein;Brunkow;Am. J. Hum. Genet.,2001
5. A 52-kb deletion in the SOST-MEOX1 intergenic region on 17q12–q21 is associated with van Buchem disease in the Dutch population;Staehling-Hampton;Am. J. Med. Genet.,2002
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2. Low Levels of Serum Sclerostin in Adult Patients With Tumor-Induced Osteomalacia Compared With X-linked Hypophosphatemia;The Journal of Clinical Endocrinology & Metabolism;2021-08-07
3. Co-deletion of Lrp5 and Lrp6 in the skeleton severely diminishes bone gain from sclerostin antibody administration;Bone;2021-02
4. Associations of serum sclerostin with bone mineral density, markers of bone metabolism and thalassaemia characteristics in adult patients with transfusion-dependent beta-thalassaemia;Annals of Medicine;2020-03-26
5. Measuring Serum Sclerostin in Egyptian Patients With Systemic Lupus Erythematosus and Evaluating Its Effect on Disease Activity;JCR: Journal of Clinical Rheumatology;2019-12-27
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