Acid sphingomyelinase (aSMase) deficiency leads to abnormal microglia behavior and disturbed retinal function-Reference-Cited by-同舟云学术

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Acid sphingomyelinase (aSMase) deficiency leads to abnormal microglia behavior and disturbed retinal function

Published:2015-08 Issue:2 Volume:464 Page:434-440
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Dannhausen Katharina,Karlstetter Marcus,Caramoy Albert,Volz Cornelia,Jägle Herbert,Liebisch Gerhard,Utermöhlen Olaf,Langmann Thomas

Funder

DFG

Hans und Marlies Stock-Foundation

Pro Retina Foundation

Marga und Walter Boll foundation

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference32 articles.

1. The pathogenesis and treatment of acid sphingomyelinase-deficient Niemann-Pick disease;Schuchman;J. Inherit. Metab. Dis.,2007

2. Sphingolipid metabolism diseases;Kolter;Biochimica Biophys0. Acta,2006

3. The metabolism of sphingomyelin. II. Evidence of an enzymatic deficiency in Niemann-Pick diseae;Brady;Proc. Natl. Acad. Sci. U S A,1966

4. Roles and regulation of secretory and lysosomal acid sphingomyelinase;Jenkins;Cell. Signal,2009

5. Principles of bioactive lipid signalling: lessons from sphingolipids;Hannun;Nat. Rev. Mol. Cell. Biol.,2008

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