Experimental autoimmune encephalomyelitis: Association with mutual regulation of RelA (p65)/NF-κB and phospho-IκB in the CNS-Reference-Cited by-同舟云学术

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Experimental autoimmune encephalomyelitis: Association with mutual regulation of RelA (p65)/NF-κB and phospho-IκB in the CNS

Published:2011-07 Issue:2 Volume:411 Page:464-470
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Hwang Insun,Ha Danbee,Ahn Ginnae,Park Eunjin,Joo Haejin,Jee Youngheun

Funder

Korea Research Foundation of the Korean Government

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference21 articles.

1. Immunological aspects of experimental allergic encephalomyelitis, multiple sclerosis;Martin;Crit. Rev. Clin. Lab. Sci.,1995

2. An essential role for NF-kappaB in preventing TNF-alpha-induced cell death;Beg;Science,1996

3. Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-kappaB;Beg;Nature,1995

4. Mutual regulation of the transcriptional activator NF-kappa B and its inhibitor, I kappa B-alpha;Brown;Proc. Natl. Acad. Sci. USA,1993

5. Effect of calpain inhibitor I, an inhibitor of the proteolysis of I kappa B, on the circulatory failure and multiple organ dysfunction caused by endotoxin in the rat;Ruetten;Br. J. Pharmacol.,1997

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