TTR exon-humanized mouse optimal for verifying new therapies for FAP-Reference-Cited by-同舟云学术

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TTR exon-humanized mouse optimal for verifying new therapies for FAP

Published:2022-04 Issue: Volume:599 Page:69-74
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Li Zhenghua,Kanazashi Hideki,Tokashiki Yoshimi,Fujikawa Rie,Okagaki Ayaka^ORCID,Katoh Sho,Kojima Kenta,Haruna Kyoko,Matsushita Naoko,Ishikawa Tomo-o,Chen Hong,Yamamura Kenichi

Funder

Japan Society for the Promotion of Science

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference24 articles.

1. A peculiar form of peripheral neuropathy; familiar atypical generalized amyloidosis with special involvement of the peripheral nerves;Andrade;Brain,1952

2. Primary structure of an amyloid prealbumin and its plasma precursor in a heredofamilial polyneuropathy of Swedish origin;Dwulet;Proc. Natl. Acad. Sci. U. S. A.,1984

3. Identification of amyloid prealbumin variant in familial amyloidotic polyneuropathy (Japanese type);Tawara;Biochem. Biophys. Res. Commun.,1983

4. Senile systemic amyloidosis;Pitkanen;Am. J. Pathol.,1984

5. Cysteine 10 is a key residue in amyloidogenesis of human transthyretin Val30Met;Takaoka;Am. J. Pathol.,2004

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Dissecting pathophysiology of a human dominantly inherited disease, familial amyloidotic polyneuropathy, by using genetically engineered mice;Frigid Zone Medicine;2022-05-01

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