The Gly–Ala repeat modulates the interaction of Epstein–Barr virus nuclear antigen-1 with cellular chromatin
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference21 articles.
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2. Epstein–Barr virus and its replication;Kieff,1996
3. EBNA-1: a protein pivotal to latent infection by Epstein–Barr virus;Leight;Rev. Med. Virol.,2000
4. A cis-acting element from the Epstein–Barr viral genome that permits stable replication of recombinant plasmids in latently infected cells;Yates;Proc. Natl. Acad. Sci. USA,1984
5. The linking regions of EBNA1 are essential for its support of replication and transcription;Mackey;Mol. Cell. Biol.,1999
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2. Regulation of EBNA1 Protein Stability by PLOD1 Lysine Hydroxylase;2022-03-29
3. Epstein–Barr virus nuclear antigen 1 interacts with regulator of chromosome condensation 1 dynamically throughout the cell cycle;Journal of General Virology;2017-02-01
4. Interaction between Basic Residues of Epstein-Barr Virus EBNA1 Protein and Cellular Chromatin Mediates Viral Plasmid Maintenance;Journal of Biological Chemistry;2013-08
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