Substrate overload: Glucose oxidation in human myotubes conquers palmitate oxidation through anaplerosis
Author:
Funder
Danish Medical Research Council
Novo Nordisk Foundation
Danish Diabetes Association
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference24 articles.
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2. Glucose plus insulin regulate fat oxidation by controlling the rate of fatty acid entry into the mitochondria;Sidossis;J. Clin. Invest,1996
3. High carbohydrate availability increases LCFA uptake and decreases LCFA oxidation in perfused muscle;Turcotte;Am. J. Physiol. Endocrinol. Metab.,2002
4. Glucose and insulin-induced inhibition of fatty acid oxidation: the glucose-fatty acid cycle reversed;Sidossis;Am. J. Physiol.,1996
5. Metabolic flexibility is conserved in diabetic myotubes;Gaster;J. Lipid Res.,2007
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1. The diabetic phenotype is preserved in myotubes established from type 2 diabetic subjects: a critical appraisal;APMIS;2018-12-14
2. A Primary Reduced TCA Flux Governs Substrate Oxidation in T2D Skeletal Muscle;Current Diabetes Reviews;2012-10-01
3. Mitochondrial mass is inversely correlated to complete lipid oxidation in human myotubes;Biochemical and Biophysical Research Communications;2011-01
4. Pyruvate carboxylase is expressed in human skeletal muscle;Biochemical and Biophysical Research Communications;2010-11
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