Mineralocorticoid receptor function in bone metabolism and its role in glucocorticoid-induced osteopenia-Reference-Cited by-同舟云学术

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Mineralocorticoid receptor function in bone metabolism and its role in glucocorticoid-induced osteopenia

Published:2014-05 Issue:3 Volume:447 Page:407-412
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Fumoto Toshio,Ishii Kiyo-aki,Ito Masako,Berger Stefan,Schütz Günther,Ikeda Kyoji

Funder

Promotion of Fundamental Studies in Health Sciences program of the National Institute of Biomedical Innovation (NIBIO)

Ministry of Health, Labor, and Welfare

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference28 articles.

1. Activation of renin–angiotensin system induces osteoporosis independently of hypertension;Asaba;J. Bone Miner. Res.,2009

2. Physiological function of the angiotensin AT1a receptor in bone remodeling;Kaneko;J. Bone Miner. Res.,2011

3. Loss of bone minerals and strength in rats with aldosteronism;Chhokar;Am. J. Physiol. Heart Circ. Physiol.,2004

4. Hyperparathyroidism and the calcium paradox of aldosteronism;Chhokar;Circulation,2005

5. Colocalization of glucocorticoid and mineralocorticoid receptors in human bone;Beavan;J. Bone Miner. Res.,2001

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