p66Shc signaling does not contribute to tubular damage induced by renal ischemia-reperfusion injury in rat
Author:
Funder
National Heart Lung and Blood Institute
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference25 articles.
1. The redox enzyme p66Shc contributes to diabetes and ischemia-induced delay in cutaneous wound healing;Fadini;Diabetes,2010
2. Role of adaptor protein p66Shc in renal pathologies;Wright;Am. J. Physiol. Ren. Physiol.,2018
3. Role of medullary blood flow in the pathogenesis of renal ischemia-reperfusion injury;Regner;Curr. Opin. Nephrol. Hypertens.,2012
4. p66shc inhibits pro-survival epidermal growth factor receptor/ERK signaling during severe oxidative stress in mouse renal proximal tubule cells;Arany;J. Biol. Chem.,2008
5. p66SHC-mediated mitochondrial dysfunction in renal proximal tubule cells during oxidative injury;Arany;Am. J. Physiol. Ren. Physiol.,2010
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