Sphingosine-1-phosphate attenuates hypoxia/reoxygenation-induced cardiomyocyte injury via a mitochondrial pathway

Author:

Ke Mengran,Tang Qiqi,Pan Ziang,Yin Yongqiang,Zhang Lizhi,Wen Ke

Funder

Chinese Natural Science Foundation

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference30 articles.

1. Essential role of the Na+-Ca2+exchanger (NCX) in glutamate-enhanced cell survival in cardiac cells exposed to hypoxia/reoxygenation;Maiolino;Sci. Rep.,2017

2. Ultrafine particulate matter increases cardiac ischemia/reperfusion injury via mitochondrial permeability;Nathan A;Transition Pore,2016

3. Myricitrin protects cardiomyocytes from hypoxia/reoxygenation injury: involvement of heat shock protein 90;Wang;Front. Pharmacol.,2017

4. A study on permeability transition pore opening and cytochrome c release from mitochondria, induced by caspase-3 in vitro;Xia;FEBS Lett.,2002

5. Distinct stages of cytochrome c release from mitochondria: evidence for a feedback amplification loop linking caspase activation to mitochondrial dysfunction in genotoxic stress induced apoptosis;Chen;Cell Death Differ.,2000

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