Transcriptional inhibition of p21WAF1/CIP1 gene (CDKN1) expression by survivin is at least partially p53-dependent: Evidence for survivin acting as a transcription factor or co-factor
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference24 articles.
1. Survivin study: what is the next wave?;Li;J. Cell. Physiol.,2003
2. Survivin study: an update of “what is the Nex wave?”;Fengzhi Li;J. Cell. Physiol.,2006
3. Survivin, cancer networks and pathway-directed drug discovery;Altieri;Nat. Rev. Cancer,2008
4. Survivin initiates cell cycle entry by the competitive interaction with Cdk4/p16(INK4a) and Cdk2/cyclin E complex activation;Suzuki;Oncogene,2000
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1. Survivin prevents the polycomb repressor complex 2 from methylating histone 3 lysine 27;iScience;2023-07
2. A RUNX-targeted gene switch-off approach modulates the BIRC5/PIF1-p21 pathway and reduces glioblastoma growth in mice;Communications Biology;2022-09-09
3. Survivin prevents the Polycomb Repressor Complex 2 from methylating Histone 3 lysine 27;2022-04-27
4. The potential roles of p53 signaling reactivation in pancreatic cancer therapy;Biochimica et Biophysica Acta (BBA) - Reviews on Cancer;2022-01
5. Chromatin binding of survivin regulates glucose metabolism in the IFN-γ producing CD4+ T cells;2021-10-05
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