Several cardiomyopathy causing mutations on tropomyosin either destabilize the active state of actomyosin or alter the binding properties of tropomyosin-Reference-Cited by-同舟云学术

Several cardiomyopathy causing mutations on tropomyosin either destabilize the active state of actomyosin or alter the binding properties of tropomyosin

Published:2011-03 Issue:1 Volume:406 Page:74-78
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Mathur Mohit C.,Chase P. Bryant,Chalovich Joseph M.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference26 articles.

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2. Hypertrophic cardiomyopathy: a systematic review;Maron;JAMA,2002

3. The genetic basis for cardiac remodeling;Ahmad;Annu. Rev. Genomics Hum. Genet.,2005

4. The genetic basis for cardiomyopathy: from mutation identification to mechanistic paradigms;Seidman;Cell,2001

5. The Δ 14 mutation of human cardiac troponin T enhances ATPase activity and alters the cooperative binding of S1-ADP to regulated actin;Gafurov;Biochemistry,2004

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