Reconstitution of defective protein trafficking rescues Long-QT syndrome in zebrafish-Reference-Cited by-同舟云学术

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Reconstitution of defective protein trafficking rescues Long-QT syndrome in zebrafish

Published:2011-05 Issue:2 Volume:408 Page:218-224
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Meder Benjamin,Scholz Eberhard P.,Hassel David,Wolff Christoph,Just Steffen,Berger Ina M.,Patzel Eva,Karle Christoph,Katus Hugo A.,Rottbauer Wolfgang

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference26 articles.

1. Chemical chaperones: a pharmacological strategy for disorders of protein folding and trafficking;Perlmutter;Pediatr. Res.,2002

2. The involvement of the Golgi apparatus in the pathogenesis of amyotrophic lateral sclerosis, Alzheimer’s disease, and ricin intoxication;Gonatas;Histochem. Cell Biol.,1998

3. Rab proteins and Rab-associated proteins: major actors in the mechanism of protein-trafficking disorders;Corbeel;Eur. J. Pediatr.,2008

4. Correctors of protein trafficking defects identified by a novel high-throughput screening assay;Carlile;Chembiochem,2007

5. Defective protein trafficking in hERG-associated hereditary long QT syndrome (LQT2): molecular mechanisms and restoration of intracellular protein processing;Thomas;Cardiovasc. Res.,2003

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