Nagarse treatment of cardiac subsarcolemmal and interfibrillar mitochondria leads to artefacts in mitochondrial protein quantification-Reference-Cited by-同舟云学术

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Nagarse treatment of cardiac subsarcolemmal and interfibrillar mitochondria leads to artefacts in mitochondrial protein quantification

Published:2018-05 Issue: Volume:91 Page:50-58
ISSN:1056-8719
Container-title:Journal of Pharmacological and Toxicological Methods
language:en
Short-container-title:Journal of Pharmacological and Toxicological Methods

Author:

Koncsos Gábor^ORCID,Varga Zoltán V.^ORCID,Baranyai Tamás,Ferdinandy Péter^ORCID,Schulz Rainer,Giricz Zoltán,Boengler Kerstin

Funder

European Association for the Study of Diabetes

National Research, Development and Innovation Office of Hungary

Hungarian Scientific Research Fund

Semmelweis University

European Cooperation in Science and Technology

Ministry Of Human Capacities

German Research Foundation

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference43 articles.

1. Acute hyperglycemia abolishes cardioprotection by remote ischemic perconditioning;Baranyai;Cardiovascular Diabetology,2015

2. Proteomic alterations of distinct mitochondrial subpopulations in the type 1 diabetic heart: Contribution of protein import dysfunction;Baseler;American Journal of Physiology. Regulatory, Integrative and Comparative Physiology,2011

3. Presence of connexin 43 in subsarcolemmal, but not in interfibrillar cardiomyocyte mitochondria;Boengler;Basic Research in Cardiology,2009

4. Mitofusin 2 tethers endoplasmic reticulum to mitochondria;de Brito;Nature,2008

5. Cardiac and skeletal muscle mitochondria have a monocarboxylate transporter MCT1;Brooks;Journal of Applied Physiology,1999

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