Removal of Multiple Arginine-Framed Trafficking Signals Overcomes Misprocessing of ΔF508 CFTR Present in Most Patients with Cystic Fibrosis
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology
Reference28 articles.
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2. Chemical chaperones correct the mutant phenotype of the delta F508 cystic fibrosis transmembrane conductance regulator protein;Brown;Cell Stress Chaperones,1996
3. Protein kinase A (PKA) still activates CFTR chloride channel after mutagenesis of all ten PKA consensus phosphorylation sites;Chang;J. Biol. Chem.,1993
4. Altered chloride ion channel kinetics associated with the ΔF508 cystic fibrosis mutation;Dalemans;Nature,1991
5. Processing of mutant cystic fibrosis transmembrane conductance regulator is temperature-sensitive;Denning;Nature,1992
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