Can we protect from malignant arrhythmias by modulation of cardiac cell-to-cell coupling?
Author:
Funder
Slovak Vega
APVV
Publisher
Elsevier BV
Subject
Cardiology and Cardiovascular Medicine
Reference47 articles.
1. Gap junction remodeling and cardiac arrhythmogenesis: cause or coincidence?;Severs;J Cell Mol Med,2001
2. Modulation of cardiac gap junction expression and arrhythmic susceptibility;Danik;Circ Res,2004
3. Reduced heterogeneous expression of Cx43 results in decreased Nav1.5 expression and reduced sodium current that accounts for arrhythmia vulnerability in conditional Cx43 knockout mice;Jansen;Heart Rhythm,2012
4. Structural substrates involved in the development of severe arrhythmias in hypertensive rat and aged guinea pig hearts;Tribulova,2002
5. Left ventricular hypertrophy: disagreements between increased left ventricular mass and ECG-LVH criteria: the effect of impaired electrical properties of myocardium;Bacharova;J Electrocardiol,2014
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