Disease-only alleles at the extreme ends of the human ZMYM3 exceptionally long 5′ UTR short tandem repeat in bipolar disorder: A pilot study

Author:

Alizadeh Fatemeh,Moharrami Tamouchin,Mousavi Negar,Yazarlou Fatemeh,Bozorgmehr AliORCID,Shahsavand Esmaeil,Delbari Ahmad,Ohadi Mina

Funder

University of Social Welfare and Rehabilitation Sciences

Publisher

Elsevier BV

Subject

Psychiatry and Mental health,Clinical Psychology

Reference22 articles.

1. Skewing of the genetic architecture at the ZMYM3 human-specific 5′ UTR short tandem repeat in schizophrenia;Alizadeh;Mol. Genet. Genomics,2018

2. Link between short tandem repeats and translation initiation site selection;Arabfard;Hum. Genomics,2018

3. Regulatory architecture of the neuronal Cacng2/Tarpγ2 gene promoter: multiple repressive domains, a polymorphic regulatory short tandem repeat, and bidirectional organization with co-regulated lncRNAs;Corney;J. Mol. Neurosci.,2018

4. Biased homozygous haplotypes across the human caveolin 1 upstream purine complex in Parkinson's disease;Darvish;J. Mol. Neurosci.,2013

5. The human RIT2 core promoter short tandem repeat predominant allele is speciesspecific in length: a selective advantage for human evolution?;Emamalizadeh;Mol. Genet. Genom.,2017

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