Interleukin-17 receptor C is essential for the pro-inflammatory pathogenicity of granulocyte-macrophage-colony-stimulating factor-producing T helper cells in experimental autoimmune uveitis-Reference-Cited by-同舟云学术

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Interleukin-17 receptor C is essential for the pro-inflammatory pathogenicity of granulocyte-macrophage-colony-stimulating factor-producing T helper cells in experimental autoimmune uveitis

Published:2023-08 Issue: Volume:390 Page:104740
ISSN:0008-8749
Container-title:Cellular Immunology
language:en
Short-container-title:Cellular Immunology

Author:

Wu Lina^ORCID,Wang Lu,Chai Xin

Publisher

Elsevier BV

Subject

Immunology

Reference31 articles.

1. Uveitis: molecular pathogenesis and emerging therapies;Egwuagu;Front. Immunol.,2021

2. Induction of antigen-specific Treg cells in treating autoimmune uveitis via bystander suppressive pathways without compromising anti-tumor immunity;Chen;EBioMedicine.,2021

3. Interleukin 12 protects from a T helper type 1-mediated autoimmune disease, experimental autoimmune uveitis, through a mechanism involving interferon gamma, nitric oxide, and apoptosis;Tarrant;J. Exp. Med.,1999

4. Critical role of interleukin (IL)-17 in inflammatory and immune disorders: an updated review of the evidence focusing in controversies;Ruiz de Morales;Autoimmun. Rev.,2020

5. A novel subset of helper T cells promotes immune responses by secreting GM-CSF;Zhang;Cell Death Differ.,2013

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