In vivo administration of recombinant BTNL2-Fc fusion protein ameliorates graft-versus-host disease in mice-Reference-Cited by-同舟云学术

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In vivo administration of recombinant BTNL2-Fc fusion protein ameliorates graft-versus-host disease in mice

Published:2019-01 Issue: Volume:335 Page:22-29
ISSN:0008-8749
Container-title:Cellular Immunology
language:en
Short-container-title:Cellular Immunology

Author:

Cui Cheng,Tian Xiaohong,Lin Yujun,Su Min,Chen Qingquan,Wang Shao-Yuan,Lai Laijun

Funder

NIH

Connecticut Regenerative Medicine Research Fund

Science and Technology Department of Liaoning Province, China

Publisher

Elsevier BV

Subject

Immunology

Reference48 articles.

1. Graft-versus-host disease;Shlomchik;Nat. Rev. Immunol.,2007

2. Graft-versus-host disease;Ferrara;Lancet,2009

3. Immunobiology of allogeneic hematopoietic stem cell transplantation;Welniak;Annu. Rev. Immunol.,2007

4. Effector CD4+ T cells, the cytokines they generate, and GVHD: something old and something new;Coghill;Blood,2011

5. Dissecting graft-versus-leukemia from graft-versus-host-disease using novel strategies;Warren;Tissue Antigens,2013

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2. Immune Checkpoint Molecules and Maternal–Fetal Immunity;Current Obstetrics and Gynecology Reports;2024-02-11

3. N-terminal ectodomain of BTNL2 inhibits T cell activation via a non-canonical interaction with its putative receptor that results in a delayed progression of DSS-induced ulcerative colitis;Molecular Immunology;2024-02

4. BTNL2 promotes colitis‐associated tumorigenesis in mice by regulating IL‐22 production;EMBO reports;2023-01-11

5. Structural Insights into N-terminal IgV Domain of BTNL2, a T Cell Inhibitory Molecule, Suggests a Non-canonical Binding Interface for Its Putative Receptors;Journal of Molecular Biology;2020-11

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