Expression of potential β-catenin targets, cyclin D1, c-Jun, c-Myc, E-cadherin, and EGFR in chemically induced hepatocellular neoplasms from B6C3F1 mice

Author:

Anna Colleen H,Iida Mari,Sills Robert C,Devereux Theodora R

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference32 articles.

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2. ras proto-oncogene activation in dichloroacetic acid-, trichloroethylene- and tetrachloroethylene-induced liver tumors in B6C3F1 mice;Anna;Carcinogenesis,1994

3. Beta-catenin mutations and protein accumulation in all hepatoblastomas examined from B6C3F1 mice treated with anthraquinone or oxazepam;Anna;Cancer Res.,2000

4. Hepatomegaly in transgenic mice expressing an oncogenic form of β-catenin;Cadoret;Cancer Res.,2001

5. The role of the cell-adhesion molecule E-cadherin as a tumour-suppressor gene;Christofori;Trends Biochem. Sci.,1999

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