From “bedside” to “bench” and back: A translational approach to studying dopamine dysfunction in schizophrenia-Reference-Cited by-同舟云学术

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From “bedside” to “bench” and back: A translational approach to studying dopamine dysfunction in schizophrenia

Published:2020-03 Issue: Volume:110 Page:174-179
ISSN:0149-7634
Container-title:Neuroscience & Biobehavioral Reviews
language:en
Short-container-title:Neuroscience & Biobehavioral Reviews

Author:

Abi-Dargham Anissa

Funder

National Institute of Mental Health (NIMH): Sylvio O. Conte Center for the Study of Dopamine Dysfunction in Schizophrenia

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Cognitive Neuroscience,Neuropsychology and Physiological Psychology

Reference47 articles.

1. Increased striatal dopamine transmission in schizophrenia: confirmation in a second cohort;Abi-Dargham;Am. J. Psychiatry,1998

2. Increased baseline occupancy of D2 receptors by dopamine in schizophrenia;Abi-Dargham;Proc. Natl. Acad. Sci. U. S. A.,2000

3. Baseline and amphetamine-stimulated dopamine activity are related in drug-naive schizophrenic subjects;Abi-Dargham;Biol. Psychiatry,2009

4. Schizophrenia is associated with elevated amphetamine-induced synaptic dopamine concentrations: evidence from a novel positron emission tomography method;Breier;Proc. Natl. Acad. Sci. U.S.A.,1997

5. FluoroDOPA PET shows the nondopaminergic as well as dopaminergic destinations of levodopa;Brown;Neurology,1999

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