Cellular and molecular neuropathology of the cuprizone mouse model: Clinical relevance for multiple sclerosis-Reference-Cited by-同舟云学术

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Cellular and molecular neuropathology of the cuprizone mouse model: Clinical relevance for multiple sclerosis

Published:2014-11 Issue: Volume:47 Page:485-505
ISSN:0149-7634
Container-title:Neuroscience & Biobehavioral Reviews
language:en
Short-container-title:Neuroscience & Biobehavioral Reviews

Author:

Praet Jelle,Guglielmetti Caroline,Berneman Zwi,Van der Linden Annemie,Ponsaerts Peter

Funder

Fund for Scientific Research-Flanders

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Cognitive Neuroscience,Neuropsychology and Physiological Psychology

Reference268 articles.

1. Preferential loss of myelin-associated glycoprotein reflects hypoxia-like white matter damage in stroke and inflammatory brain diseases;Aboul-Enein;J. Neuropathol. Exp. Neurol.,2003

2. 17beta-estradiol and progesterone prevent cuprizone provoked demyelination of corpus callosum in male mice;Acs;Glia,2009

3. Selective ultrastructural vulnerability in the cuprizone-induced experimental demyelination;Acs;Ideggyogy Sz.,2012

4. Distribution of oligodendrocyte loss and mitochondrial toxicity in the cuprizone-induced experimental demyelination model;Acs;J. Neuroimmunol.,2013

5. Remyelination after cuprizone-induced demyelination in the rat is stimulated by apotransferrin;Adamo;Exp. Neurol.,2006

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3. Targeting the PAC1 receptor mitigates degradation of myelin and synaptic markers and diminishes locomotor deficits in the cuprizone demyelination model;Journal of Neurochemistry;2024-08-08

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5. Nalfurafine promotes myelination in vitro and facilitates recovery from cuprizone + rapamycin‐induced demyelination in mice;Glia;2024-06-20

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